Vaccinazioni per l’infanzia ed
dei vaccini = Autismo vedi: PDF - dott.
raccolta di dati medico-scientifici sui gravi danni dei Vaccini
Vaccini ed Autismo = Bibliografie che comprovano il
Altra Bibliografia,157 studi scientifici, che confermano il
nesso Vaccini= Autismo !
Sentenza 2012 - Trib. Rimini su
Commento NdR: sulla sentenza di Rimini: vaccini =
fatto il Giudice del Tribunale di Rimini (Italia) a
sentenziare in quel modo, perche' egli non si e'
lasciato influenzare dalle FALSITA' del
Ministero della "salute" (che e'
stato da noi
informato sui Danni dei vaccini dal 1996 e
se ne sta zitto.....assieme a
tutti gli altri "enti"....) fino agli ordini
dei medici......tutti al servizio di
Big Pharma !
- vedi lo studio del dott.:
In CINA dopo le campagne vaccinali esplode
l'Autismo ! - Maggio 2016
VERISSIMO, ma non solo l'autismo....ma una
innumerevole sequela di altre
e non solo dai Vaccini:
USA, Giugno 2013 - AUTISMO = 1 bambino
autistico su 26, non come era nel 2010, 1
su 80 ....
I Tribunali anche USA, confermano tranquillamente che il
MMR causa l'autismo. Austin (USA) - 27 Luglio 2013
Dopo decenni di appassionato dibattito, per i genitori che
probabilmente hanno perso i ripetuti ricorsi richiesti dalle
aziende farmaceutiche e governi, che i vaccini infatti causano
Per i genitori interessati alla ricerca della verità, vale la
pena ricordare che le stesse persone che possiedono le aziende
farmaceutiche di tutto il mondo possono anche possedere agenzie
di stampa americane.
La Ricerca di informazioni prive di propaganda è stata fino ad
ora molto difficile.
Ma Whiteout Press non è qui per sostenere o contrastare i
vaccini. Siamo qui per portare i lettori la notizia che è il
tema e’ in black-out, cover-up e censurato dalle autorita’Sanitarie
conferma che i Vaccini possono produrre l'
La prova della FRODE del
per le cause dei
- CONFESSIONE di un alto dirigente
CDC, davanti al Congresso US
Gli esperti di
CDC, hanno spesso
Conflitti di interesse - 1
CDC e Conflitti
di interesse - 2
CDC e Conflitti
di interesse - 3
anche per i vaccini +
anche per la FDA
Davvero inquietante !
Questo medico il Dott.
Andrew Moulden è MORTO (probabilmente
assassinato) in modo inspiegabile nel
novembre 2013 al età di 49, subito dopo
aver pubblicato Le SUE RICERCHE che
DIMOSTRANO il DANNO CAUSATO dai VACCINI,
RICONOSCIBILI SOLO da un SEMPLICE ESAME
Parlamentari pagati dalle Lobbies ? -
Roma Ott. 2013
L'intervista a un assistente di un Senatore
che svelerebbe i traffici illeciti tra
CONFESSA la FRODE e le FALSIFICAZIONI sugli
studi della correlazione VACCINO=AUTISMO
QUI, tutti gli studi, ricerche, interviste, citate nel
documentario sull'autismo dai Vaccini Vaxxed
0007EK - Sex: M -
Age: 5 -
and Postnatal History:
Uncomplicated prenatal and postnatal history. Birth weight 8
lbs.,apgars 9 and 9.
Easily met all early milestones.
Parents report precocious language skills.
At 10 months he was talking with phrases “oh, there it is.”
At 12 months there was a major and obvious reversal in behavior.
Speech, social interaction, and laughter began to fade away
He began toe
walking, lost eye contact, grew inattentive, and developed repetitive
Mother with 8 dental amalgams, no fish consumption. Infant
received thimerosal in vaccines, but unable to calculate exposure at
this time. At 3 years of
age 8 amalgam fillings were placed with an initial improvement in
behavior for 3 weeks, then a decline to a level much worse than before
the dental work with progressive decline.
Prior to chelation non-detectable, 12-27-99.
DMPS IM + oral DMSA/EDTA and DMSA/EDTA supp. (unspecified
41 mcg / g creatine of urinary mercury.
supp. 250mg bid were used 3 x week, every other week subsequent to
provocation testing. Oral
DMSA provocation for urinary Hg pending.
Multiple dietary and secretin infusions are concurrent to the
DMPS/DMSA chelation, but mother is firmly convinced that the latter are
contributing to excellent behavioral and somatic gains.
Improvement in eye contact within 2 days of DMSA is evident.
Improvement in speech, sociability and playing with toys are seen
consistently right after DMSA and are reported to be on a gradual upward
trend. A full sentence was
uttered on or about 3-1-00.
addition to the above case studies, we have collected preliminary data
on three autistic children who have not undergone chelation. These
children also exhibit elevated levels of mercury.
on Non-Chelated ASD Children
level and source of sample
metal hair analysis
analysis 1.2ug/g (ref. <0.4)
PRBC 18.4 (ref <9)
analysis 1.8 ppm
observations from these case studies deserve mention.
One is that all of the children experienced a regressive form of
autism. Other findings are that (i) low levels of mercury in hair may be
associated with large amounts of mercury excretion on provocation and
(ii) initial levels of provoked mercury may not be as high as subsequent
ones. Mercury in the hair will only reflect a current or recent exposure
of approximately one year or the body's active detoxification of
mercury. This was evident
in a child with non-detectable levels of mercury in the hair and
positive levels on provocation.
the case studies there is also a trend of higher numbers for mercury in
younger children (20 month hair sample of 4.8 ppm and 2 ½ year hair
sample of 5.6 ppm). This may be related to the fact that the testing was
performed closer to the time of exposure.
Hair levels of mercury greater than 5.0 ppm are considered
diagnostic for mercury poisoning (Applied Toxicology, 1992).
Among the majority of these case studies much more modest
elevations of mercury, if detected at all, were associated with high
levels of provoked mercury.
are no standards for provoked levels of mercury in children in the
context of behavioral disorders. Therefore,
we surveyed a large number of physicians treating adults with chronic
health problems diagnosed as secondary to mercury. These clinicians
advise that tolerable limits may vary according to the general health of
the patient and associated health problems.
All consulted agreed that in adults excretion of 50 mcg of
mercury per gm creatine after intravenous DMPS challenge is worrisome.
We submit that the concern level for children should be even more
stringent. High levels of
mercury are demonstrated in some children without a history of fish
consumption, amalgam burden, or known environmental exposure, suggesting
the role of vaccines as a contribution to body burden.
families who submitted these case histories wanted to tell their stories
because their children are noticeably improved after treatment for
mercury. Whether this improvement was sudden or gradual, the parents
are convinced that lessening the mercury and heavy metal burden has
helped their child. They
ask us to request support for much needed research in this area.
reasonable is it to claim that the most common form of autism, where
there is normal development and then regression, could be caused by
There are several reasons to believe that this process has
Criteria Are Met
literature demonstrates that mercury can induce autism-spectrum traits,
and this association extends to mercury’s localization within specific
to address “the totality of the syndrome” (Bailey et al, 1996), we
have shown that every major characteristic of autism has been exhibited
in at least several cases of documented mercury poisoning, and that
every major area of biological and neurological impairment implicated in
ASD has been observed with Hg exposure.
Recently, government-directed studies have revealed that the
amount of mercury given to infants receiving vaccinations exceeds safety
levels. The timing of mercury administration via vaccines coincides with
the onset of autistic symptoms. Case reports of autisticchildren with
measurable mercury levels in hair, blood, and urine indicate a history
of mercury exposure along with inadequate detoxification. Thus the
standard criteria for a diagnosis of mercury poisoning in autism, as
outlined at the beginning of thispaper, are met. In other words, mercury toxicity is a significant
contributing factor or primary etiological factor in many or most cases
Form Would be Expected, Implicates Vaccinal Thimerosal
manifested in mercury poisoning are diverse and vary by the interaction
of variables such as type of mercury, age of patient, method of
exposure, and so forth. Thus,
although it could be argued that in all the thousands of cases of past
Hg poisonings, no instance of autism could be found,such an argument
fails to take into account the possibility of unique expression.
It would be comparable to saying that, because in all the cases
of Minamata disease no instance of acrodynia could be found, then
acrodynia could not be caused by mercury poisoning.
Since there are no case reports or systematic studies in the
literature of the effects of intermittent bolus doses of injected
ethylmercury on “susceptible” infants and toddlers, it would be
reasonable to expect that symptoms arising from this form of mercury
poisoning would present as a novel disease.
In fact, given the high neurotoxicity of organic mercury, its
known psychological effects, and the age at which it has been given in
vaccines, it would almost be a given that the “novel disease” would
present as a neurodevelopmental disorder like autism.
the fact that autism meets the diagnostic criteria for mercury
poisoning, yet has never been described as a mercury-induced disease,
requires that the disorder must arise from a mode of mercury
administration which has not been studied before. This would rule out
other known sources of Hg like fish consumption or occupational mercury
hazards, as these have been well characterized. It is possible that
another under-investigated mercury route, such as maternal Hg exposures
(e.g., from vaccinations, thimerosal-containing RhoGam injections during
pregnancy, or dental fillings) or infant exposures to
thimerosal-containing eardrops or eyedrops, might be a factor, and this
cannot be ruled out.
is a precedent for large scale, undetected mercury poisoning of infants
and toddlers in the syndrome that came to be known as acrodynia or pink
disease. For over 50 years,
tens of thousands of children suffered the bewildering, debilitating,
and often life-long effects of this disease before its mercury etiology
was established, as Ann Dally relates in The Rise and Fall of Pink
Disease (1997, excerpts):
is a serious disease that was common, at least in children’s clinics,
during the first half of the present (20th) century. Reports abound of
children too miserable to acknowledge their mothers, such as the child
who kept repeating, “I am so sad.” One unhappy mother was quoted as
saying,“My child behaves like a mad dog.” In most cases the
condition improved spontaneously, but was often regarded as chronic.
Mortality varied from 5.5% to 33.3% and was usually about 7%. Most
physicians who speculated on the causes of pink disease believed in
either the infective or the nutritional theory. No one seems to have
suggested that it might be due to poisoning. It was a tradition to
advise student doctors to treat cases of difficult teething with the
mercury powders that were eventually to be revealed as the cause of the
disease. The ill-effects of mercury on the mouth had been known at least
since the time of Paraclesus, but it was not until 1922 that the
pediatrician, John Zahorsky, commented on the similarity between pink
disease and mercury poisoning. He dismissed rather than pursued his new
idea of possible mercury poisoning and suggested a theory that was more
in tune with current fashion. Most doctors, even those skilled in the
use of calomel, associated mercury poisoning with adults (syphilis,
industrial poisoning, hatters shakes) rather than with infants. By 1935
the disease was seen in every children’s out-patient clinic.
mystery began to be solved in 1945 by Dr. Josef Warkany, of the
Cincinnati Children’s Hospital. He and his assistant found large
amounts of mercury in the urine of a child with pink disease. They did
not publish their findings until 1948, but it is noteworthy that the
news seems not to have spread through the small and tightly knit
pediatric world, where everyone knew everyone else. It was probably
because the idea was unfashionable and contrary to the conventional
wisdom. The theory that mercury poisoning caused pink disease was
gradually accepted, but against resistance, particularly by older men
and those in powerful positions. Mercury was withdrawn from most
teething powders after 1954, initially through voluntary action by the
manufacturers because of adverse publicity and probably in the hope of
avoiding statutory prohibition. Pink disease almost disappeared. Later
in the decade the theory was widely accepted and soon pink disease was
no longer part of the usual pediatric out-patient clinic."
like acrodynia before it, autism may in fact be “just another”
epidemic of mercury poisoning, this time caused by childhood vaccinal
mercury rather than infant teething powders.
Preventing Earlier Discovery Are Removed
priorities and methods of research experts in the autism and mercury
fields have prevented the association between mercurialism and ASD to be
recognized until recently.
effects on humans of mercury-containing medicinals and home remedies
used to be studied quite regularly by medical researchers (Warkany and
Hubbard, 1953); but since,aside from vaccinal thimerosal, such products
have declined dramatically in number since the 1950s and 1960s, most
mercury researchers today focus on biochemical studies or environmental
sources like fish and coal plants. Some mercury experts seem surprised
to learn that Hg is present in infant vaccines (authors’ personal
experience), and as recently as 1997, when the EPA released its massive
review of extant mercury research, vaccines were not even mentioned as a
potential source. Thus it is not surprising that mercury experts have
never investigated thimerosal as they have, say, contaminated whale meat
consumption in the Faroes Islands or Hg exposure among Amazonian
it is not surprising that neither mercury experts nor autism
professionals have ever investigated autism as a possible disease of
Since its discovery by Kanner, autism has been
characterized in almost exclusively psychological terms. The
descriptions have been such that the symptoms would be essentially
unrecognizable as manifestations of poisoning to any mercury expert not
looking closely. A perfect
example is Kanner himself, who recorded feeding problems and vomiting in
infants and concluded: “Our patients, anxious to keep the outside
world away, indicated this by the refusal of food.” Bruno Bettleheim,
who dominated autism discourse in the 1950s and 1960s and blamed the
entire disorder on “refrigerator mothers” who forced the withdrawal
of the child, asserted, "the source of the anxiety is not an
organic impairment but the child's evaluation of his life as being
utterly destructive" (1967, reported in ARI Newsletter). In 1987,
Robert Sternberg would propose a“unified theoretical perspective on
autism” by defining the disorder in terms of a “triarchic theory of
intelligence,” and in the same publication Lorna Wing and Anthony
Attwood would write:
young autistic children will stand in a dejected posture, with tears
streaming down their faces, as if they suddenly felt their helplessness
in theface of a world they cannot understand."
as recently as 1995, a typical slate of articles in the dominant Journal
of Autism and Developmental Disorders (April 1995) would consist of
eight psychological pieces (example: “Generativity in the Play of
Young People with Autism”) and one biomedical one (on biopterin). Thus
biomedical research in autism existed, but it was mostly relegated to
the margins as psychology held center stage, and the symptomatic
characteristics of autism continued to be presentedin accord with
the latter part of the 1990s, the situation on both sides changed.
Congressional mandate led to the public quantification of the cumulative
amount of mercury in vaccines, raising interest in understanding its
effects. Parent organizations like CAN and NAAR, working with the NIH
and other researchers, engineered an autism research agenda which is
more heavily focused on underlying physiological mechanisms of the
disease. With parents already suspecting a vaccine-autism link, the
environment was right for investigations focused on the link between
vaccinal mercury and autism.
& SOCIETAL IMPLICATIONS
NIH (1999, web site) estimates that there are nearly half a million
Americans who suffer from autism, a devastating, debilitating, and
lifelong disorder. Given the role of thimerosal as a major contributing factor
in ASD, basic and clinical research efforts should be focused on
understanding how mercury leads to autism in susceptible individuals and
on finding effective methods to address the resulting Hg damage. Such
research might focus on the following areas, with others undoubtedly
still to be identified:
methods which will work across all body tissues and especially the
brain. The current standard chelators – DMPS and DMSA - appear unable
to cross the blood-brain barrier. Other promising but less studied
chelators like alpha lipoic acid can cross the bbb (Fuchs et al, 1997)
and should be studied in autism.
to induce immunity to Hg and which might possibly reverse the Th2 shift
or IFNg expression which mercury causes. The work of Hu and colleagues
suggests that Hg can cause an immune reaction in any individual, but
some are protected by a counteractive immunosuppressive response, and
Warkany and Hubbard have pointed out that individuals who are
Hg-sensitive can later become “immune”. It may be possible to
engineer these responses in autistic individuals through careful
which might reverse Na-Si transporter blockage in the intestines and
kidney, thereby normalizing sulfate absorption.
to eliminate the Hg-induced epileptiform activities found in the
majority of autistic children, as outlined by LeWine et al.
cell applications in autism to repair brain damage that occurred during
pointed out by David Hartman (1998), mercury’s ability to cause a wide
range of common psychiatric disturbances should be considered in their
diagnosis, and it might also be productive in developing hypotheses
about and designing research studies for these other disorders. The disorders might include depression, OCD, dementia, anxiety,
ADHD/ADD, Tourette's, and schizophrenia. Mercury may play arole in the etiology of some cases of these
investigating mercury’s wide ranging effects upon neurobiological
processes may lead to a quicker understanding of the organic etiologies
in these other diseases which are now seen with increasing frequency.
compliance with the recommended vaccine schedule is a governmental,
medical, and societal goal, since “vaccines save lives” (CDC).
Our goal is not to negatively impact childhood immunization
rates. Instead, we have
been careful to distinguish between thimerosal and vaccines.
Thimerosal is not a vaccine; it is a preservative.
Except for trace amounts, vaccines without
currently available for all routinely recommended immunizations for
children under 6 years (Institute for Vaccine Safety, 1999).
Furthermore, it is possible to remove mercury from existing
products. Merck, for example, delivered and received FDA approval for a
thimerosal-free Hepatitis B vaccine in a record-breaking two months from
the time the FDA publicly encouraged manufacturers to develop
thimerosal-free alternatives (Pless, 1999; Merck, 1999).
Thus, any issues being raised here are related to how vaccine
programs are run, not with vaccines themselves.
issues, of course, are: (i) first, how thimerosal was allowed to remain
a component of the immunization program, even after 1953 when Warkany
and Hubbard specifically named vaccinal mercury as a possible factor in
acrodynia, or 1982 when the FDA issued a notice singling out thimerosal
as especially neurotoxic as well as ineffective as a preservative
(Federal Register, 1982); and (ii) second, why thimerosal remains in
over 30 vaccine products today (FDA, 1999), and why the FDA, as of March
2000, has only "encouraged" rather than required the vaccine
manufacturers to remove the thimerosal (William Egan personal
the CDC has stated that no adverse effects from thimerosal have been
found other than hypersensitivity reactions, the sad fact is there have
been no direct studies on the long term effects of intermittent bolus
doses of ethylmercury injected in infants and toddlers.
As Altman and Bland have aptly demonstrated (1995), “absence of
evidence is not evidence of absence.”
lapses in vaccine program oversight suggest that vaccine safety studies
need to be bolstered. Current
practice is to track adverse reactions only if they occur within one
month of the vaccination. The
experience with mercury clearly shows that an adverse event may not
manifest for months if not years. Studies
on adverse reactions must involve long term tracking of patients; they
should investigate the impact of multiple injections as well as compare
reactions to vaccines with and without various additives; and sample
sizes need to be large enough to include especially sensitive groups.
Finally, the FDA should require manufacturers to remove all remaining
thimerosal from their vaccines immediately, so that another child is not
lost to this terrible disease.
would like to thank the following people for their important
contributions to this article: Amy Rosenberg, Ayda Halker, Andrew
Cutler, Edie Davis, Merri Adler-Ross (Bergen County Community Service
Program, Hackensack, NJ), Mark Maxon, Thomas Marchie, Ramone Martinas,
Michael DiPrete, Nancy Gallo, David Patel and Paramus Library, Reference
Desk (Paramus NJ). -
Autismo dai VACCINI
Autismo - La prova dei
Danni dei Vaccini
Bibliografia su Autismo dai vaccini +
Danni dei vaccini +
Amish senza autismo
perche' NON vaccinano +
1.000 studi sui Danni dei Vaccini
molta importanza hanno anche i cibi assunti non
adatti al gruppo sanguigno del soggetto.
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