|
MALATTIE
CAUSATE
dai VACCINI e dalle SOSTANZE ivi
PRESENTI
Fra il
1940 ed il 1955 si venne a sapere che i
bambini che
erano stati
vaccinati recentemente
per la pertosse
erano soggetti a contrarre la
polio paralitica in misura
crescente rispetto a coloro che
non erano
stati
vaccinati per questa malattia.
Durante l'epidemia di
polio in Minnesota, nel 1946 ben 85 bambini furono
colpiti dalla malattia: 35 di essi avevano ricevuto di
recente il vaccino per la pertosse (da 5 a 19 giorni prima)
e gli arti nei quali era stata fatta l'iniezione
risultarono paralizzati nel 58% dei bambini che avevano
contratto la
polio.
Nel 1949, il rischio di contrarre la
polio paralitica
infantile era 4 volte maggiore
nei soggetti che avevano ricevuto il vaccino DP (Difterite -
Pertosse) entro le 6
settimane precedenti, alla
falsamente detta, esposizione al
virus: questi dati
furono raccolti comparando il gruppo dei
bambini
vaccinati con un gruppo di controllo
NON vaccinato.
Nel
1953, una ricerca effettuata su un'epidemia di polio
verificatasi in alcune isole del Pacifico dimostrò come
i bambini che avevano ricevuto iniezioni settimanali di
una soluzione di mercurio, arsenico e bismuto per
combattere una infestazione di spirochete avessero
successivamente sviluppato la polio in un rapporto 10:1
rispetto a quelli che non avevano ricevuto il
trattamento.
Nel
1954, uno studio intitolato "Fattori scatenanti e
localizzanti nella poliomielite", condotto da Trueta e
Hodes e pubblicato sulla rivista
The Lancet, delinea le
ricerche condotte a partire dall'inizio del 1900 sui
diversi fattori che sembravano aumentare la gravità
delle poliomieliti, ovvero che determinavano la loro
localizzazione in aree specifiche del sistema nervoso.
Già negli anni '20 i ricercatori ritenevano che il
virus
della polio potesse migrare attraverso l'organismo
attraverso il sistema della circolazione sanguigna.
Questo breve accenno indusse Trueta e Hodes a condurre i
loro studi ipotizzando che il fattore che determina la
gravità e la localizzazione della polio poteva in
qualche modo trasferirsi dai vasi sanguigni nel sistema
nervoso, aumentando la
permeabilità della barriera fra
sangue e cervello, permettendo in tal modo al virus
della polio di accedere con maggiore facilità sia al
cervello che al
sistema nervoso centrale.
Gli esperimenti con la formalina e le altre
sostanze
chimiche utilizzate nei
vaccini dimostrano la
congestione dei vasi sanguigni nelle zone corrispondenti
alla localizzazione della paralisi: queste ricerche ed i
lavori successivi hanno confermato la proprietà di
queste sostanze chimiche di aumentare la
gravità della
malattia e le probabilità di
decesso, in funzione anche
del batterio o virus che viene iniettato
contemporaneamente con il vaccino.
Uno
studio successivo, del 1954, da parte del Consiglio
delle Ricerche Mediche della Gran Bretagna, rivelò che
il vaccino DP, specialmente quello ottenuto per
precipitazione grazie ai sali e
idrossidi di alluminio,
predisponeva i bambini a
contrarre la polio paralitica.
Nel 1957, l'eminente biologo Dubos
dimostrò che
l'iniezione di vaccino per la pertosse o di micobatterio
ucciso nelle cavie di laboratorio infettate alcuni mesi
prima con piccole dosi dello stesso batterio
causava una
moltiplicazione batterica esplosiva: in altre parole, il
vaccino per la pertosse è in
grado di accelerare delle
forme di infezione latente verso infezioni acute
vere e proprie.
Dal momento che non si ritiene di esaminare in modo
completo coloro che vengono vaccinati, non è possibile
individuare qualsiasi forma di infezione latente che può
essere in tal modo risvegliata dal vaccino stesso, dalle
sostanze chimiche e dagli antibiotici che li
accompagnano.
IMPORTANTE da
ricordare:
Una delle caratteristiche dei
danni
cellulari dei
vaccini e’ quella di
inibire e/o alterare la formazione di proteine essenziali
allo sviluppo regolare dei tessuti ed organi di un
bambino, e fra questa una si chiama
Eps8 e gioca un ruolo fondamentale nei processi di
memoria e apprendimento.
Milano 2013 (I) - A svelarne i meccanismi molecolari, un
team di ricercatori italiani dell’Università degli Studi di
Milano, Istituto di Neuroscienze del Cnr e Humanitas. Lo
studio, pubblicato su Embo Journal, aprirà la strada a nuovi
percorsi di cura per affrontare i gravi problemi legati ad
autismo e
ritardo mentale e dello sviluppo.
Vaccini obbligatori o no, tutti quanti sono da
sempre sbandierati come utili e sicuri anche e non solo
dal
Ministero della salute, ma esiste una sentenza ben
precisa (ignorata da tutti…) che mostra come tali
distinzioni fra vaccini obbligatori e non, NON possono
sussistere - vedi:
Sentenza Corte Costituzionale
Le famiglie dei
danneggiati da vaccino dal canto loro sottolineano
“lo stato di abbandono in cui sono state lasciate dalle
cosiddette “istituzioni che dovrebbero tutelare”,
infatti “tutelano” ma solo i
fatturati di
Big Pharma….". +
Corruzione per i Vaccini
Ricerca medica sul
collegamento fra Vaccini e malattie
autoimmuni !
http://info.cmsri.org/the-driven-researcher-blog/first-medical-textbook-devoted-to-research-linking-vaccines-to-autoimmunity
Danni dei Farmaci e Vaccini, eminenti medici/ricercatori
indipendenti lo confermano
Uno studio in
inglese dimostra, che bambini NON
vaccinati sono più sani che i bambini
vaccinati
http://www.naturalblaze.com/2014/02/studies-prove-without-doubt-that.html
Video su: I BAMBINI
NON
VACCINATI SONO molto PIU' SANI di quelli
VACCINATI !
Nel 1992 I.A.S. ha condotto un sondaggio
sulla salute e la vaccinazione dei
bambini della Nuova Zelanda ed ha
trovato solide prove scientifiche che I
BAMBINI NON VACCINATI sono PIU' SANI dei
loro coetanei vaccinati !
VACCINI OMICIDIO
di MASSA
Un Rapporto ufficiale
rivela che l'uno per cento dei bambini americani, hanno
l'autismo
e
che esso e’ in crescita esponenziale.
I
Centers for Disease Control (CDC)
hanno pubblicato un nuovo rapporto che mostra che uno su
ogni 110 bambini in America oggi hanno una qualche forma
di autismo e cio’ nel 2006,
nel 2013 1 su 25.
Con una stima di tre quarti di un milione di bambini e’
immessa da qualche parte, nello spettro autistico; il
gruppo di pressione per i Bambini autistici, “Autism
Speaks”, chiede al governo federale di aumentare gli
sforzi di ricerca per combattere la malattia.
Il rapporto, pubblicato sulla morbilità e mortalità su Weekly
Report, illustra un aumento del 57 per cento nei casi
di autismo tra il 2002 e il 2006.
Un incredibile aumento del 600 per cento la malattia si
è verificato nel corso degli ultimi due decenni ,
affligge più spesso i maschi rispetto alle femmine.
Circa uno su ogni 70 ragazzi, attualmente ha una qualche
forma di autismo.
Scienziati e ricercatori riconoscono che il tasso di
autismo è in aumento in modo significativo e che
qualcosa deve essere fatto per affrontare il problema.
Uno studio del 2007 della
Harvard School of Public Health, osserva che 35
miliardi dollari sono spesi ogni anno per trattare le
persone che soffrono di autismo, e che i bambini sono il
segmento in più rapida crescita delle persone che
vengono diagnosticati con la malattia.
I bambini sono spesso diagnosticati con autismo di età
compresa tra sei e otto anni, un'età che molti credono
sia troppo tardi, poiché la malattia è di solito
osservabile negli anni precedenti . Allo stato attuale,
le compagnie di assicurazione in genere non coprono
trattamento dell'autismo.
Autism Speaks ha adottato l'approccio convenzionale,
sollecitando un aumento dei finanziamenti per la ricerca
a livello federale.
Invece di indagare su un approccio preventivo, gli
avvocati stanno implorando il governo a rafforzare i
finanziamenti in modo che i bambini possono essere
diagnosticati in precedenza e ricevere il trattamento
per i loro sintomi in età precoce.
Commenti di Mike Adams, il Ranger della Salute
Per ogni bambino che viene diagnosticato l'autismo, ci
sono genitori che si chiedono, "ha fatto i vaccini e
causano questo" ?
I tassi di autismo sono saliti alle stelle negli ultimi
due decenni, provocando un numero crescente di genitori
a chiedersi cosa c'è dietro la preoccupante tendenza.
L'autismo è chiaramente qualcosa a che fare con problemi
neurologici, ed è del tutto ragionevole sospettare che i
prodotti chimici infiammatori
utilizzati nei vaccini come adiuvanti possono
svolgere un ruolo importante nel causare l'autismo.
Ci possono essere altre cause, anche, come l'esposizione
a pesticidi,
metalli pesanti, solventi chimici e sostanze
chimiche tossiche utilizzate nei prodotti per la cura
personale.
Raramente ho visto bambini che non sono bagnate da
shampoo tossici, lozioni per la pelle, creme solari e
saponi.
L'ingrediente antibatterico in sapone è una sostanza
chimica neurotossica, e ancor più neurotossine si
trovano nella catena alimentare.
L'autismo potrebbe essere semplicemente il risultato di
un carico cumulativo di prodotti chimici neurotossici
provenienti da più fonti:
Vaccini, prodotti di
consumo, alimenti, medicinali, detergenti per la casa e
altro ancora.
By Mike Adams – By NaturalNews
Source link:
http://www.naturalnews.com/028310_autism_neurotoxic_chemicals.htm
vedi:
Danni da
Vaccino PDF (english)
>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>
800,000 vaccine
induced injuries (English)
"My
"agenda" is to tell the truth. Like the fact that, according to
Centers for Disease Control (CDC) statistics, as many as 800,000 vaccine
induced injuries have occurred every year in the United States since
1990.
"--Leonard Horowitz
The
US Federal Government's National Vaccine Injury Compensation Program (NVICP)
has paid out over 724.4 million dollars to parents of vaccine injured and
killed children, in taxpayer dollars.
The
NVICP has received over 5000 petitions since 1988, including over 700 for
vaccine-related deaths, and there are still over 2800 total death and
injury cases pending that may take years to resolve (NVICP, Health
Resources and Services Administration).
"The
carnage caused by vaccinations has become so immense, and the outcry from
the grieving parents so intense, that the government has set up a national
compensation program in order to smooth everything over and to protect
drug companies and doctors from law suits. You, of course, pay for this
insurance for drug companies and doctors by the cost being added to the
price of the vaccines. In 1982, the vaccines cost $23 per child. By
1992, the cost had risen to $244--an increase of over 1,000 percent !
This
devastation of our children by our own doctors and public-health
departments has been so colossal that over $249 million has been awarded
for vaccine-caused injuries and deaths, and the program is now
bankrupt.Thousands of cases are pending that will recieve nothing--the pay
window has been slammed shut.
Even if you got lucky and recieved "compensation," will that
make up for your child's permanent paralysis ("Guillain-Barre
syndrome"), blindness ("idiopathic macular degeneration"),
mental deficiency ("learning disorder"), or incoordination
("tardive dyskinesia")? Will all those phoney diagnoses used to
cover up the real diagnosis help ?
Will a million dollars make everything
okay ?
Ten million ?"---William Douglas MD
Hassan
W, Oldham R. Reiter’s syndrome and reactive arthritis in health care
workers after vaccination. British Medical Journal 1994; 309: 94
MECHANISMS
OF VACCINATION SEQUELAE
by
Teresa Binstock Researcher in Developmental and Behavioral Neuroanatomy
http://www.jorsm.com/~binstock/vacc-let.htm
At
Whale
"Role
of Immunogenetics in the Diagnosis of Postvaccinal CNS Pathology,"
Massimo Montinari, et al., Department of Pediatric Surgery, University of
Bari, Italy, presented May 9, 1996 (text available
http://www.healthy.net/library/articles/coulter/biochem.htm
): after thirty children were found to have signs of central nervous
system and genetic damage following vaccination, the authors remark,
"A study of the disease associated with genes of the HLA system has
shown that this genetic complex can be responsible for am particular
genetic susceptibility, predisposing to various diseases characterized
predominantly by immune-system pathogenesis… results indicate that
autoimmune pathology is more frequent in countries where vaccination is
more widespread.."
[A fuller description of this study will be
found in "The attenuated virus--infectious or not?" below.]
(Pediatric
Bulletin,
http://home.coqui.net/myrna/virus.htm
).
"Disease
caused by Haemophilus influenzae type b in the immediate period after
homologous immunization: immunologic investigation" (Pediatrics, vol.
85, number 4 part 2, April 1990, pp. 698-704):
"One concern with the
use of [current HIB vaccines] was the suggestion that the incidence of
invasive disease caused by H influenzae type b in the immediate period
after immunization might be increased; this idea was supported by evidence
from several sources."
In one case-controlled study, 4 children were
hospitalized for invasive disease within 1 week of immunization; the rate
of invasive disease was 6.4 times greater than the background rate in
unvaccinated children.
"Neurologic
complications associated with oral poliovirus vaccine and genomic
variability of the vaccine strains after multiplication in humans,"
Acta Virologica, vol. 42, number 3, June 1998, pp. 187-94: The oral
poliovirus vaccine (OPV) sometimes occasions paralytic poliomyelitis in
vaccine recipients and their susceptible contacts. Molecular biology
studies of polioviruses from these patients demonstrate genomic
modifications known or suspected to increase neurovirulence.
The same
genomic modifications have been identified in strains isolated from
non-symptomatic vaccinees.
Other neurologic complications such as
meningitis, encephalitis, convulsions, transverse myelitis and
Guillain-Barre Syndrome have also been associated with this vaccine.
"Transmission
of vaccine strain varicella-zoster virus from a healthy adult with
vaccine-associated rash to susceptible household contacts" (Journal
of Infectious Disease, vol. 176, no. 4, October 1997, pp. 1072-5):
Twelve
days after receiving an investigational Oka strain live attenuated
varicella vaccine, a 38-year-old healthy woman developed a rash consisting
of 30 scattered lesions.
Sixteen days later, her two children also
developed a rash. Varicella-zoster DNA obtained from the skin lesions was
determined to be the vaccine type. "This case ocuments transmission
of varicella vaccine type virus from a healthy vaccinee to susceptible
household contacts…ongoing studies will define the frequency of this
transmission."
"Live
Virus Vaccines, High-Dose Steroids Don't Mix" (Pediatric News, cited
November 28, 1998, [email protected], 10:49 a.m.): Dr. Larry K. Pickering, a
member of the American Academy of Pediatrics'
"Red Book Committee,"
was quoted following a meeting at the University of South Dakota, saying
children receiving more than 2 mg/kg per day of systemic glucocorticoids
should not be given live virus vaccines, due to the risk of disseminated
infection from the vaccines. Killed virus vaccines do not present the same
risk. [Note: steroids such as prednisone partially suppress the immune
system.]
"Acute
encephalopathy followed by permanent brain injury or death associated with
further attenuated measles vaccines: a review of claims submitted to the
National Vaccine Injury Compensation Program," Pediatrics, vol. 101,
no. 3, Part 1, March 1998; pages 383-387:
This study details cases wherein
48 children, ages 10 to 49 months, who had been so affected. Eight
children died, and the remainder had mental regression and retardation,
chronic seizures, motor and sensory deficits, and movement disorders.
"CONCLUSIONS: This clustering suggests that a causal relationship
between measles vaccine and encephalopathy may exist as a rare
complication of measles immunization."
[Note regarding rarity: A huge
number of vaccine reactions are never reported, and most of the thousands
of vaccine injuries which are reported do not meet the current, very
narrow VAERS/FDA criteria (a very few specific symptoms must occur within
a very short timespan, in order for symptoms to be considered
vaccine-related), and thus are not reported as vaccine-injury cases by
government tabulators. Serious vaccine complications thus are said to be
"rare" in quoted statistics. If independent research proves that
the measles vaccine and PDD/autism are causally related, this kind of
vaccine damage will inflate by thousands the cases of vaccine damage now
on record.
This tally, then, may be inflated further by the number of ADD/ADHD-diagnosed
children with inflammatory bowel disorders, per the Georgetown University
study cited I "Wakefield," below.]
"Measles-Mumps-Rubella
(MMR) Vaccine as a Potential Cause of Encephalitis (Brain Inflammation) in
Children," Harold E. Buttram, MD, Townsend Letters, December 1997
(available
at
http://www.mercola.com/issue5.htm
).
T.
Zecca, D. Grafino, et al., University of Medicine and Dentistry, New
Jersey and Children's Hospital of New Jersey, Newark, "Elevated
rubeola [measles] titers in autistic children linked to MMR vaccine"
(abstract submitted to the National Institutes of Health, 1997-8;
text
available at:
http://webpages.netlink.co.nz/~ias/mmraut1.htm
:
Rubeola (measles) titers were compared in autistic and normal children.
Children diagnosed with autism revealed "a three fold increase"
in their rubeola titers over expected normal range. "A Wilcoxon
Kruskal Wallas test comparing 13 rubeola titers from normal children
reveals a statistically significant P-value of 0.0050." The authors
note that neurological sequelae following MMR are widely reported: "MMR
therefore may play a role in the pathogenesis of Autism.
The elevated titers of anti-measles antibodies in Autistic children may signify a
chronic activation of the immune system against this neurotropic
virus."
"Characterisation
of poxviruses from sporadic human infections" (South African Medical
Journal, vol. 72, no. 12, December 19, 1987, pp. 846-8): An orthopoxvirus
was isolated from…a man in Natal who died in coma…
Analysis of the
viral DNA showed that it was a vaccinia virus, more closely related to the
virus of South African smallpox vaccine than to other [natural] vaccinia
viruses. DNA analysis also showed that an orthopoxvirus isolated from a
sporadic case of severe pustular rash in Nigeria was a vaccinia virus
closely related to the smallpox vaccine virus used there… [It was]
suggested that some natural transmission of the virus had occurred…originat[ing]
from the use of smallpox vaccine. No similar cases have been detected
since smallpox vaccination was discontinued."
"Vaccinia
virus persistence in a child against the background of immune deficiency"
(J. Hyg. Epidemiol. Microbiol. Immunol., vol. 30, no. 2, 1986, pp.
177-83):
" A young girl, vaccinated against smallpox 6 years before[,]
suffered from a persistent vaccinia virus infection and a congenital skin
disesase, i.e. epidermolysis bullosa. The virus was isolated from skin
lesions at the vaccination site and remote sites and repeatedly from the
blood… Examination of the child did not show any quantitative immune
deficiency…
The possible genesis of the virus persistence and the role
of the virus in the clinical course of the disease are discussed." (A
selected Medline [National Library of Medicine] "MESH" subject
tracing for this report is "Smallpox Vaccine--adverse effects.")
"Polymerase
chain reaction detection of the hemagglutinin gene from an attenuated
measles vaccine strain in the peripheral mononuclear cells of children
with autoimmune hepatitis," Archives of Virology volume 141, 1996,
pages 877-884: "The measles virus is known to be persistent in
patients with subacute sclerosing panencephalitis (SSPE) and measles
inclusion body encephalitis (MIBE).
Since the introduction of measles
vaccines, vaccine-associated SSPE has increased in the USA. Therefore, we
should pay attention to SSPE after inoculation with measles vaccine,
despite the decrease in the incidence of [wild] measles."
"The
African polio vaccine-acquired immune deficiency syndrome connection"
(Medical Hypotheses, vol. 48, no. 5, May 1997, pp. 367-74):
"Seroepidemiological,
clinical and molecular findings suggest that the acquired immune
deficiency syndrome virus Human Immunodeficiency Virus-1* was introduced
into the human species at the the (late 1950s) and in the geographic area
(Zaire) in which millions of Africans were vaccinated with attenuated
poliomyelitis virus strains that were produced in kidney tissue obtained
from monkeys. …it is reasonable to suspect that a then non-detectable
monkey virus with human-1-like properties was unknowingly cocultured with
the attenuated poliovirus and subsequently administered to the vaccinees.
The possibility of such a polio vaccine-acquired immune deficiency
syndrome connection is a reminder of the unpredictable danger of
artifically crossing natural species-barriers in biomedical laboratories"
[*bold text capitals added].
"The
origin of HIV-1, the AIDS virus" (Medical Hypotheses, vol. 41, no. 4,
October 1993, pp. 289-99): "a substantial case is presented that
HIV-1 is a natural recombinant of Bovine Leukemia Virus (BLV) and Visna
Virus. This natural recombinant may have been inadvertently transferred to
humans through the Intensified Smallpox Eradication Program conducted in
sub-Saharan Africa in the late 1960s and most of the 1970s."
"Simian
cytomegalovirus-related stealth virus isolated from the cerebrospinal
fluid of a patient with bipolar psychosis and acute encephalopathy"
(Pathobiology
, vo. 64, no. 2, 1996, pp. 64-6): a cytopathic 'stealth' virus was
cultured from the cerebrospinal fluid of this patient, who developed a
severe encephalopathy leading to a vegetative state.
DNA sequencing of a
polymerase chain reaction-amplified product from infected cultures
revealed kinship to the African green monkey simian cytomegalovirus.
Disorders
of the ear
Blood
disorders
"Thrombocytopenic
purpura as adverse reaction to recombinant hepatitis B vaccine" (Archives
of Disease in Childhood, vol. 78, no. 3, March 1998, pp. 273-4): Three
cases of [auto]immune thrombocytopenic purpura after the first dose of
recombinant hepatitis B vaccine occurred in infants under six months of
age. There were no other possible causes; defect in platelet production
was excluded in two children. Antiplatelet antibodies were present.
The
babies were treated with corticosteroids.
Hepatitis
"Polymerase
chain reaction detection of the hemagglutinin gene from an attenuated
measles vaccine strain in the peripheral mononuclear cells of children
with autoimmune hepatitis,"
Archives of Virology volume 141, 1996,
pages 877-884: Four pediatric and two adults patients with autoimmune
hepatitis were tested and followed in this study. Twelve healthy children
served as controls, who had either been infected with measles or
vaccinated with an attenuated measles vaccine in the past. All controls
were negative for measles virus except a recent (two week) vaccinee. Of
the hepatitis patients, all were positive for measles virus—the children
with vaccine-strain measles virus, and the adults with different strains.
Conclusion: "our results demonstrated that children with autoimmune
hepatitis can have persistence of the vaccine strain in vivo for many
years after vaccination [abstract, page 877]." The authors state that
the persistence of the measles virus might play some role in the pathology
of autoimmune hepatitis, but further studies are needed to prove this
hypothesis (page 883).
Also
in "Polymerase," the authors observe that high levels of serum
antibodies to measles virus have been reported in patients with autoimmune
hepatitis (p. 877). References add systemic lupus erythematosus and
infectious mononucleosis to the tally of autoimmune diseases with
connections to measles (pages 883-4).
[Note: high antibody titers of
measles and rubella are also associated with autism.]
Some
provocative quotes, page 882:
"Apparently,
the attenuated vaccine is also capable of persisting, like sporadic wild
strains, in certain immune diseases. The measles virus is known to be
persistent in patients with subacute sclerosing panencephalitis (SSPE) and
measles inclusion body encephalitis (MIBE).
Since the introduction of
measles vaccines, vaccine-associated SSPE has increased in the USA.
Therefore, we should pay attention to SSPE after inoculation with measles
vaccine, despite the decrease in the incidence of [wild] measles."
[Note:
the following study did not broach the subject of vaccine involvement in
diseases; rather it serves to point out the relationship of viral
presences to disease.] …
Department of Virology, University of Helsinki,
Finland, "Very high measles and rubella virus antibody titres
associated with hepatitis, systemic lupus erythematosus, and infectious
mononucleosis"
(The Lancet, vol. 1, February 9, 1974, pp. 194-7): In
patients without preceding rubella or measles infection, "raised
levels of viral antibodies were a constant finding in two repeated
analyses" of hepatitis patients.
The authors felt that "it is
conceivable that rubella and/or measles infections or reinfections may
cause acute hepatitis and persist in some individuals…such aberrant
virus infection might be responsible for some clinical manifestations….."
Chronic virus infection could not be excluded as an important factor in
these diseases.
Inflammatory
and autoimmune bowel disease
"Paramyxovirus
infections in childhood and subsequent inflammatory bowel disease"
(Gastroenterology,
vol. 116, no. 4, April 1999, pp. 796-803): "Measles virus has been
implicated in the etiology of both inflammatory bowel diseases (IBDs),
Crohn's disease and ulcerative colitis… Mumps infection before age 2
years was a risk for ulcerative colitis… Measles and mumps infections in
the same year of life were significantly associated with ulcerative
colitis and Crohn's disease…but not with IDDM…
Atypical paramyxovirus
infections in childhood may be risk factors for later I[nflammatory] B[owel]
D[isease]" [Notes: measles-mumps-rubella vaccine is usually given
around the age of 16 months. When vaccine viruses induce infection, it is
often atypical in character].
Lupus,
multiple sclerosis and rheumatoid arthritis
Abstract:
autoimmune diseases are becoming increasingly common. The majority seem to
have viral associations.
"Vaccine-induced
autoimmunity" (Journal of Autoimmunity, vol. 9, no. 6, December 1996,
pp. 699-703): the authors summarize of case reports attributing autoimmune
diseases and autoimmune phenomena to vaccines, and suggest possible
mechanisms by which the two could be related.
"The subject is
complicated," they say, "by the fact that one vaccine may cause
more than one autoimmune phenomenon, and a particular immune process may
be caused by more than one vaccine. Furthermore, vaccines differ in their
pathogenic influence on the immune system... The subject of the
vaccine-autoimmunity relationship is still obscure; reports have been
rare, [and] no laboratory experimentation on this topic has been
undertaken....."
(Oddly, the authors state that the benefits of
vaccination outweigh the risks of disease, but given the authors'
contentions that vaccines can cause one or more types of autoimmune
disease, that reports are few and research non-existent, this statement is
unsupported. Further, they conclude that "laborious clinical and
laboratory studies should be initiated in order to evaluate the ..subject.")
C.
M. Poser, Harvard Medical School, "The pathogenesis of multiple
sclerosis. Additional considerations" (Journal of Neurological
Science, vol. 115, April 1993, Supplement pp. S3-15):
"Multiple
sclerosis is acquired as a systemic "trait" by individuals who
are genetically susceptible…It develops as the result of an antigenic
challenge by a viral protein, either from a viral infection or a
vaccination."
"Multiple
sclerosis and infectious childhood diseases"
(Neuroepidemiology, vol.
17, no. 3, 1998, pp. 154-60): multiple sclerosis patients studied had had
measles, mumps, and varicella (chicken pox) infections at a later age than
healthy controls. "These results are compatible with the hypothesis
that the risk of developing multiple sclerosis may be associated with
acquiring certain infectious childhood diseases at a later state in
comparison to normal controls."
[Early vaccination for these diseases,
therefore, may predispose vaccinees to MS, as immunity from vaccinations
frequently wanes in the years following early childhood vaccination (unlike
immunity to natural infection). In the event of such a vaccine failure,
natural infection may occur at a later age.]
"Chronic
arthritis after rubella vaccination" (Clin. Infectious Disease, vol.
15, no. 2, August 1992, pp. 307-312. After reviewing a wide range of
information sources, The Institute of Medicine, Washington, DC, found a
causal relationship between rubella vaccination and chronic arthritis in
adult women.
--for
lupus, see <<cognitive disorders>> below--
Parasthesias/paralytic
and muscular diseases
"Drug
Points: Transverse Myelitis After Measles, Mumps, and Rubella
Vaccine," BMJ [British Medical Journal], vol. 311 (7002), August 12,
1995, p. 422: a twenty-year-old man was vaccinated against rubella with
the MMR vaccine. Five days later he developed fever, malaise, sore throat,
and a transient, upper-body rash.
Within the next two weeks, he developed
an ascending paraesthesia. He was hospitalized on developing a rapidly
progressive flaccid paraplegia. Serological tests showed a significant
rise in rubella antibodies. Postvaccination transverse myelitis was
diagnosed.
"Poliovirus
vaccine options" (American Family Physician, vol. 59, no. 1, January
1, 1999, pp. 113-8, 125-6): "Of 142 confirmed cases of paralytic
poliomyelitis reported in the United States from 1980-1996, 134 were
classified as vaccine-associated paralytic poliomyelitis (VAPP). Persons
with VAPP have a disabling illness….."
"Demonstration
of specific antineuronal nuclear antibodies in sera of patients with
myasthenia gravis" (Neurology, vol. 24, no. 7, July 1974, pp. 680-3).
Other
disorders of the brain and nervous system
Abstract:
Vijendra K. Singh and others have found a significant association between
autoimmune processes in autistic patients and viral presences--in
particular, anti-myelin basic protein (anti-brain) antibodies, along with
high titers of specific viruses. In this regard, see also "Demonstration
of specific antineuronal nuclear antibodies," above, and the
description of T. Zecca's report, "Elevated rubeola [measles] titers
in autistic children linked to MMR vaccine," above.
<<seizure
disorders>>
"Autistic
subjects with comorbid epilepsy: a possible association with viral
infections" (Child Psychiatry and Human Development, vo. 29, no. 3,
Spring 1998, pp. 245-51): Data covering a 30-year period was examined in
Israel.
The annual birth pattern of 290 autistic subjects with comorbid
epilepsy fit the seasonality of viral meningitis. "These findings
support the role of viral C[entral] N[ervous] S[ystem] infections in the
causality of this disorder."
"Neurologic
complications after vaccination against diphtheria, tetanus and whooping
cough (Cesk. Pediatr., vol. 47, no. 2, February 1992, pp. 122-4): Both in
children free from neurological disease and in children with neurological
disease the most frequent type of complications from DTP vaccination were
"encephalopathies and febrile attacks as a consequence of metabolic
and toxic changes following vaccination."
Persisting neurological
disorders were, in the majority, epileptic in character.
"Vaccination
against whooping-cough. Efficacy versus risks," The Lancet, vol. 1,
January 29, 1977, pp. 234-7: "Adverse reactions and neurotoxicity
following vaccination was strongly related to pertussis vaccine in 79 of
160 cases studied. A shock reaction and cerebral disturbance was seen, in
most of these cases followed by
convulsions,
hyperkinesis, and severe mental defect. The authors conclude, "It
seems likely that most adverse reactions are unreported and that many are
overlooked…existing provisions, national and international, for
epidemiological surveillance and evaluation are inadequate. The claim by
official bodies that the risks of whooping-cough exceed those of
vaccination is questionable, at least in the U.K."
O.
Tonz and S. Bajc, "Convulsions or status epilepticus in 11 infants
after pertussis vaccination" (Schweiz. Med. Wochenschr., vol. 110,
no. 51, December 20, 1980, pp. 1965-71): In three of 11 cases, grand mal
epilepsy persisted and two children developed infantile epileptic
encephalopathy (Lennox Syndrome).
"The following conclusions are
drawn from these observations: 1) In view of the usually benign course of
whooping cough today, current vaccination is hardly satisfactory.
Improvement of the available vaccines is an urgent necessity… 2) Parents whould be better informed about the risks involved in pertussis
vaccination. 3) Booster inoculations should be abandoned. 4) Health
authorities should decide whether the current pertussis vaccination
program should be abandoned. 5) Complications following vaccination should
be registered….."
<<behavior
and movement disorders >>
"A
controlled study of serum anti-locus ceruleus antibodies in REM sleep
behavior disorder" (Sleep, vol. 20, no. 5, May 1997, pp. 349-51):
"The newly identified association of human nonnarcoleptic rapid eye
movement (REM) sleep behavior disorder (RBD) with human leukocyte antigen
(HLA) DQwl class II genes raises the possibility that RBD may arise from
autoimmune mechanisms."
[The
following reports are not vaccine-specific; rather they serve to underline
one of the possible conditions resulting from altered permeability of, or
damage to the intestine, as occurs in association with measles and other
viruses. Note: strep-type bacteria are among those which can translocate
from the gut; these have been implicated in cases of Obsessive-Compulsive
Disorder and Tourette Syndrome.] "Bacterial translocation from the
gastrointestinal tract" (Trends in Microbiology, vol. 3, no. 4, April
1995, pp. 149-54): Viable indigenous bacteria from the gastrointestinal
tract can migrate to other sites within the body, such as the
mesenteric-lymph-node complex, liver, spleen, and bloodstream. Three
mechanisms support bacterial translocation: intestinal bacterial
overgrowth, deficiencies in host immune defenses and increased
permeability or damage to the intestinal mucosal barrier.
"Case
study: a new infection-triggered, autoimmune subtype of pediatric OCD and
Tourette's syndrome"
(Journal of the American Academy of Child and
Adolescent Psychiatry, vol. 34, no. 3, March 1995, pp. 307-11): the
authors hypothesize that infections with group A beta-hemolytic
streptococci, among other bacterial agents, may trigger autoimmune
responses that cause or exacerbate some cases of childhood-onset
obsessive-compulsive disorder (OCD) or tic disorders including Tourette's
Syndrome. In this study, four boys aged 10 to 14 years presented with OCD
or Tourette's Syndrome in the moderate to very severe range. Two had
evidence of recent group A beta-hemolytic streptococci infections, and the
others had histories of recent viral illnesses.
"Speculations
on antineuronal antibody-mediated neuropsychiatric disorders of childhood"
(Pediatrics, vol. 93, no. 2, February 1994, pp. 323-6): "Several
converging lines of evidence suggest that some behavioral and neurological
abnormalities of childhood may be mediated through antineuronal antibodies.
These antineuronal antibodies appear to arise in response to group A
[beta]-hemolytic streptococcal (GABHS) infections and to cross-react with
cells within the central nervous system (CNS). Based on clinical
observations of children with Sydenham's chorea, Tourette's syndrome (TS),
and/or obsessive-compulsive disorder (OCD), we hypothesize that
neuroimmunological dysfunction secondary to antineuronal antibodies may
result in behavioral disturbances, such as anxiety, emotional lability,
obsessive compulsive symptoms, hyperactivity, and sleep disturbances, and
neurological abnormalities, such as motor and phonic tics, ballismus,
chorea, and choreiform movements."
"Antineuronal
antibodies: tics and obsessive-compulsive symptoms" (Journal of
Developmental and Behavioral Pediatrics, vol. 15, no. 6, December 1994,
pp. 421-5): 19 or 38 cases from an ongoing study of childhood
neurodevelopmental disordershad existing or previously docuemnted OCS [OCD]
and attention-deficit hyperactivity disorder (ADHD), with or without
concomitant tics. 19 controls had ADHD, but no tics or OCS. Evidence was
found of basal ganglia involvement in OCS, and a generalized central
nervous system response [to infection] was suggested.
"Bipolar
disorders, dystonia, and compulsion after dysfunction of the cerebellum,
dentatorubrothalamic tract, and substantia nigra" (Biological
Psychiatry, vol. 40, no. 8, October 1996, pp. 726-30): the mechanism of
the legions was not abstracted in this report; however, after focal
cerebellar circuit lesions, these disorders presented in three of fifteen
subjects.
"Antineuronal
antibodies in movement disorders" (Pediatrics, vol. 92, no. 1, July
1993, pp. 39-43): 24 children with recent-onset movement disorders (Tourette
Syndrome, motor and/or vocal tics, chorea, and choreiform movements) as
well as ADHD, behavior disorders, or learning disabilities were studied.
The authors concluded that their data strongly suggests an association
between antecedent group A beta-streptococcal infection and serum
antineuronal antibodies, which may, in turn, be linked to childhood
movement disorders.
"Antibodies
to human caudate nucleus neurons in Huntington's chorea" (Journal of
Clinical Investigation, vol. 59, no. 5, May 1977, pp. 922-32): IgG
antibodies against nervous system components were detected in patients
afflicted with Huntington's and Parkinson's Diseases, as well as in
asymptomatic spouses of patients.
"These data may support an
environmental or infectious factor somehow involved in the ultimate
expression of HD."
[This
report is not vaccine-specific, but underlines a radical shift in thinking
about cerebral palsy and a variety of other neurological impairments--i.e.,
to an infectious etiology.] "Infections may underlie cerebral palsy"
(Science News, vol. 154, no. 16, October 17, 1998, p. 244; available at
http://www.sciencenews.org/sn_arc98/10_17_98/fob1.htm
:
"Most doctors have believed that cerebral palsy--a form of brain
damage that impairs movements--results from a difficult birth… While
asphyxia may indeed be a cause of cerbral palsy, a new study provides
evidence that the brain damage might often arise from some other…assault
on an unborn child. Molecular clues now lead to inflammatory infection as
a possible culprit, says Karein B. Nelson, a pediatric neurologist at the
National Institute of Neurological Disorders and Stroke in Bethesday, MD.
"
A study was performed by Nelson and colleagues which compared blood from
normal and CP infants: the team found that all the stricken children harbored greater concentrations of substances indicating immune activation.
In some of the children, indications of autoimmunity were seen as well. (Study
citation: "Neonatal cytokines and coagulation factors in children
with cerebral palsy," Annals of Neurology, vol. 44, October 1998, p.
665.)
"Increased
prevalence of antibrain antibodies in the sera from schizophrenic patients"
(Schizophrenia Research, vol. 14, no. 1, December 1994, pp. 15-22); "Antibodies
to brain tissue in sera of schizophrenic patients-preliminary findings"
(European Archives of Psychiatry and Clinical Neuroscience, vol. 242, no.
5, 1993, pp. 314-7):
Antibrain antibodies have been found in the sera of
schizophrenic patients, but not in normal controls.
These seem to be
directed against brain centers affected in schizophrenia.
<<cognitive
disorders >>
"Serum
autoantibodies to brain in Landau-Kleffner variant, autism, and other
neurologic disorders" (Journal of Pediatrics, vol. 134, no. 5, May
1999, pp. 607-613): "Etiologically unexplained disorders of language
and social development have often been reported to improve in patients
treated with immune-modulating regimens.
Here we determined…children
with L[andau] K[leffner] S[ydrome] V[ariant] and A[utistic] S[pectrum] D[isorder]
have a greater frequency of serum antibodies to brain endothelial cells
and to nuclei than children with non-neurologic illnesses or healthy
children. The presence of these antibodies raises the possibility that
autoimmunity plays a role in the pathogenesis of language and social
developmental abnormalities in a subset of children with these disorders.
"Characteristics
of antineuronal antibodies in systemic lupus erythematosus patients with
and without central nervous system involvement: the role of mycobacterial
cross-reacting antigens" (Israeli Journal of Medical Science, vol.
26, no. 7, July 1990, pp. 367-73): indirect immunofluorescence of human
brain tissue sections revealed, in thirteen of sixteen patients, high
antineuronal antibody titers.
Competition assays showed that the binding
of the antineuronal antibodies was blocked by mycobacterial glycolipids
and bovine brain extracts.
"This
finding suggests an additional link between mycobacterial infection and
SLE."
"An
immunological approach to dementia in the elderly" (Age and Ageing,
vol. 5, no. 3, August 1976, pp. 164-70): Immunofluorescence studies showed
"an excess of antineuronal reactivity and a fall in antinuclear
antibody in females with senile dementia."
Alteration
of human genetic code
Abstract:
viruses are able to infiltrate cells, inserting their genetic material
into them. Indications have been found of changes to human genetic
characteristics as a result of viral invasion.
"Role
of Immunogenetics in the Diagnosis of Postvaccinal CNS Pathology,"
Department of Pediatric Surgery, University of Bari, Italy, presented May
9, 1996 (text available
http://www.healthy.net/library/articles/coulter/biochem.htm):
7
initially, thirty young children were tested and followed who showed the
first symptoms of CNS pathology with or immediately after vaccination with
polio, DT, measles, DPT, anti-tuberculosis, or Hepatitis-B vaccines.
Immediate reactions to the vaccines included convulsions, high fever, or
diarrhea with or immediately after vaccination. Among the post-vaccinal
symptoms were encephalopathies, food allergies, constipation, diarrhea,
and other central nervous system pathology.
Diagnoses applied to subjects
after vaccination and before this study were epilepsy of various types; epileptigenic encephalopathy, autism, West Syndrome, and Angelman's
Syndrome.
There
were no genetic or metabolic anomalies revealed during testing which might
have explained the CNS symptoms.
The viral encephalopathies which
presented with or following vaccination were not due to transplacental
viral infection. EEGs after initial symptoms were negative in 92 percent.
Following vaccination and CNS symptoms, serologic investigations for
herpes viruses were positive in all cases for IgG. IgG for Epstein-Barr
virus and cytomegalovirus were estimated to be positive in 73.8/71.4
percent respectively, herpes simplex in 47.6 percent, and varicella zoster
in 21.4 percent of patients. 73.3 percent of subjects showed an increase
in the HLA-A3 and HLA-DR7 antigens as compared with the Italian population
at large.
The
authors found and describe, in this paper, biochemical markers of vaccine
damage (e.g., changes in inherited HLA type). They also point out that
most vaccines contain thimerosal, a toxic substance associated with
neurologic and gastrointestinal symptoms.
The fact that post-vaccinal
pathologies of the central nervous system are often not thoroughly
investigated occasioned this study. Additional cases are under study to
better define the possible association of HLA A3 and/or HLA DR7 with this
CNS pathology following vaccination.
"New
Genetic Study Points Way for Vaccine Reaction Research/Novel Genetic
Clinical Marker Found in Blood of Gulfwar Vets" (Press release,
National Vaccine Information Center/PR Newswire, Washington, D.C., May 3,
1999, 5:48 p.m.; original source is Clinical and Diagnostic Laboratory
Immunology, May 1999):
A three year study funded and conducted by the
Chronic Illness Research Foundation in collaboration with the University
of Michigan School of Medicine found abnormal RNA in the blood of 50
percent of sick Gulf War veterans, indicating that chromosomal damage had
occurred. This genetic material was not found in any of the healthy
controls.
Damage to chromosome 22q11.2 has been linked in other published
studies to autoimmune diseases such as juvenile rheumatoid arthritis and
other illnesses like multiple myeloma cancer. The discovery of RNA in the
cell-free fractions of blood is an anomaly, as it is not normally present
in serum. RNA can exist outside the cell only if it is protected, as RNA
viruses can.
Gulf War soldiers were given 17 different viral and bacterial
vaccines, including experimental anthrax and botulinum toxoid vaccines.
Experimental drugs were also given and [in veterans actually deployed to
the Gulf] there were exposures to pesticides, low-level chemical warfare
agents, low-level radiation, toxic combustion products, etc. The resultant
symptoms are similar to those of vaccine-damaged children.
Dr. Howard B. Urnovitz, microbiologist and Science Director of the Chronic Illness
Research Foundation, interpreted findings to indicate that certain
genotypes may be particularly at risk for sustaining chromosomal damage
after exposure to toxic events; ways to identify and prescreen for
individuals who may be at high risk for chromosomal damage should be found.
Many
thanks to Laura J. Ruede who supplied the text and study details above
http://lib.tcu.edu/www/staff/lruede/autvacc
Vaccine
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Syphilis
from smallpox vaccination (1880)
A
Smallpox Vaccine Disaster Record (1855--1880)
>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>>
Massachusetts Deaths Caused
by Measles: 1860 to 1970
101. See
generally Thomas F. Burke, Lawyers, Lawsuits, and
LegaL Rights: The Battlle Over Litigation In American
Society 142-70 (2002).
102. See generally id.
103. Restatement of the Law 2d, Torts, § 402A comment k
(American Law Institute 1965).
104. See Givens v. Lederle, 556 F.2d 1341 (5th
Cir. 1977); Reyes v. Wyeth Laboratories, Inc., 498 F.2d
1264 (5th Cir. 1974); Davis v. Wyeth Laboratories, 399
F.2d 121 (9th Cir. 1968).
105. See Burke, supra note 101, at 144
(citing Edward W. Kitch, Vaccines and Product
Liability: A Case of Contagious Litigation,
Regulation, May/June 1985, at 13).
106. See id. at 163.
107. See Toner v. Lederle, 828 F.2d 510 (1987).
108. See Johnson v. Amer. Cyanamid Co., 718 P.2d
1318 (Kan. 1986).
109. See Burke, supra note 101, at 144.
110. Statement of Robert B. Johnson, president, Lederle
Laboratories Division, American Cyanamid, House
Subcommittee on Health and the Environment, Vaccine
Injury Compensation, Sept. 10, 1984, cited in
Burke, supra note 101, at 250 n.30.
111. Id.
112. See U.S. General Accounting Office,
Childhood Vaccines: Ensuring an Adequate Supply Poses
Continuing Challenges 5 (Sept. 2002), available
at
http://www.gao.gov/new.items/d02987.pdf;
Jim Copland, Liable to Infection Flu Vaccine in Short
Supply Partly Because of Trial Lawyers and "Tort Tax,"
Dallas Morning News, Dec. 14, 2003, available
at
http://www.manhattan-institute.org/html/_dmn-liable_to_infection_flu.htm.
113. See Burke, supra note 101, at 150.
114. See Testimony of James Mason, director,
Division of Immunization of the Center for Prevention
Services, Centers for Disease Control, Public Health
Service, House Subcommittee on Health and the
Environment, Vaccine Injury Compensation, Dec.
19, 1984, cited in Burke, supra note 101, at 250
n.29.
115. See National Childhood Vaccine Injury Act of
1986, 42 U.S.C. § 300 (2004).
116. See Burke, supra note 101, at 160-61.
117. See id.
118. See id. at 163 (citing U.S. Dept. of Health
& Human Services).
119. See id. at 161.
120. See id.
121. See id.
122. See id. at 163.
123. See id.
124. See Centers for Disease Control, Lyme
Disease—United States, 2001–2002, at
http://www.cdc.gov/mmwr/preview/mmwrhtml/mm5317a4.htm;
Centers for Disease Control, Lyme Disease— United
States, 1999, at
http://www.cdc.gov/mmwr/preview/mmwrhtml/mm5010a1.htm.
For an example of class-action litigation over LYMErix,
see the practice area under the firm website for
the law firm Sheller Ludwig & Badley, at
http://sheller.com/PracticeAreas.asp.
125. See Kenneth Todar, Lyme Disease,
Todar's Online TextBook of Bacteriology, at
http://textbookofbacteriology.net/Lyme.html
("Between 1996 and 2001 the number was level at about
17,000 new cases per year, but increased by nearly 7,000
cases in 2002.").
126. See William Tucker, La Grippe of the
Trial Lawyers, Weekly Standard, Oct. 25, 2004,
available at
http://www.weeklystandard.com/Content/Public/Articles/000/000/004/793dgqvs.asp;
Richard Kent Zimmerman, American Academy of Family
Physicians, Lowering the Age for Routine Influenza
Vaccination to 50 Years: AAFP Leads the Nation in
Influenza Vaccine Policy, Nov. 1, 1999 (citing W. W.
Williams et al., Immunization Policies and Vaccine
Coverage among Adults: The Risk for Missed Opportunities,
108 Ann. Intern. Med. 616, 616-25 (1988)).
127. See News Release, U.S. Department of Health
& Human Services, HHS Says Supply of Flu Vaccines,
Medicines Will Help Keep People Safe During Coming Flu
Season, Oct. 19, 2004, available at
http://www.hhs.gov/news/press/2004pres/20041019a.html.
128. See Copland, supra note 112.
129. See U.S. Food and Drug Administration,
Thimerosal in Vaccines, at
http://www.fda.gov/cber/vaccine/thimerosal.htm
("At the initial National Vaccine Advisory
Committee-sponsored meeting on thimerosal in 1999,
concerns were expressed that infants may lack the
ability to eliminate mercury.").
130. See Politicizing Vaccines, Wall St. J., Nov.
18, 2002, available at
http://www.ph.ucla.edu/epi/bioter/politicizingvaccines.html.
131. U.S. Food and Drug Administration, supra
note 129.
132. Id.
133. See Copland, supra note 112.
134. See American Academy of Pediatrics,
Recommended Childhood and Adolescent Immunization
Schedule, United States, 2005, at
http://www.cispimmunize.org/IZSchedule.pdf.
135. See World Health Organization, Statement on
Thiomersal, Aug. 2003, at
http://www.who.int/vaccine_safety/topics/thiomersal/statement200308/en/index.html
("The Global Advisory Committee on Vaccine Safety.....concluded that the latest pharmacokinetic and
developmental studies do not support concerns over
safety of thiomersal (ethyl mercury) in vaccines. The
Committee has concluded, and advises accordingly, that
there is no reason on grounds of safety to change
current immunization practices with thiomersal-containing
vaccines, since the benefits outweigh any unproven
risks.").
136. See Lawsuits Indicate Growing National Movement
to Hold Vaccine Producers Liable for Mercury Poisoning
Epidemic, Idaho Observer, Aug. 2001, available at
http://proliberty.com/observer/20010807.htm.
137. See Copland, supra
note 112.
Tratto da:
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